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Deborah Kaye Bell (Allen)

Birthplace: Old St. Joseph Hospital, Joliet, IL, United States
Immediate Family:

Daughter of Hugh Albert Allen, Jr. and Joyce Marie Sebring
Wife of Private
Mother of Private and Dawnna Marie Mullen
Sister of Private User; Private and Private User
Half sister of Private and Private

Last Updated:
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Immediate Family

About Debbie Bell (Allen)

I've been married 44 yrs have 2 wonderful children, 2 wonderful step children, 11 Grandchildren, and 9 Great grandchildren.

I was not brought up near my cousins or grandparents, on either side. There were brief stays with a relative while my dad looked for new lodging during our moves to and from military School at Fort Bliss in El Paso, TX. However, we never stayed for very long so I think not knowing my family brought out this great need for family and so I started my search in the late 1980s. Dad took over the search when I went back to work full time, then at his passing in 2011 I found a box her called "Family Search", as I was going through his things, and the search was passed back to me. It became a full time hobby when I became disabled in 2014. Since then I have dedicated a good deal of my time to family research and in the process discovered cousins that I didn't know I had, reconnected with cousins, and have learned a lot about where I came from.

My paternal grandmother, Arivella Baer, married Hugh Albert Allen Sr and together they had 5 children. Unfortunately Hugh passed away from a rare spinal tumor just a few months before my father, Hugh Albert Allen Jr. was born. Instantly my grandmother became a single mother of 5. My dad always described her as a hard working mom. She worked at the local dime store in Alma, MI, had a very large garden, and always made homemade bread, jellies, pickles, and a whole lot more. But in spite of all their efforts they still relied on the government care packages to make ends meet. Arivella married Roy Harms when my dad was about 12 years old, this match was a soulmate type of match and they lived happily together until their deaths.

Hugh, my dad, married Joyce Franks in Sept of 1958 and in November my brother Bob was born, almost exactly one year later I was born, and in 1961 my brother Ron was born. In 1962 Dad and mom separated, mom moved back to Riverdale and in n May of 1963 my sister Tammie was born. She was thought, by my dad, to be a child of her 2nd family. However, DNA has proven that she is a full sibling.

Besides Tammie, my mom had two other children, Elizabeth (Betty) and Eric Sebring, and I was lucky enough to meet all three in the summer of 1975 when I went and stayed with them in Kissimmee, Melbourne, and finally St. Cloud, Florida.

Mom's father is Harold Franks b. 1908 d. 1947, her mother is Myrtle Vanderrmark .b.1907 d.1978 were married 24 Nov 1928 at Pine Dale, Gratiot, Michigan, USA. They had 7 children, 4 boys and 3 girls, one that died at birth.

Ftdna kit #69425

Haplogroup: U5a2c4

HVR1 Mutations: A16129G, T16187C, C16189T, C16192T, T16223C, G16230A, C16256T, C16270T, T16278C, C16311T, C16519T, G16526A

HVR2 Mutations: C146T, C152T, C195T, A247G, A493G, 522.1A, 522.2C, 315.1C

My current FTdna Origins 4/3/2021:

  • Europe 100%
  • Western Europe
    • England, Wales, and Scotland 61%
  • Central Europe 32%
  • Southern Europe
    • Iberian Peninsula 7%
  • Middle East:
    • Anatolia, Armenia, & Mesopotamia <1%

My Heritage ethnicity results 4/3/2021

    • Irish, Scottish, and Welsh 39.6%
    • Scandinavian 35.5%
      • Sweden (Halland and Skåne)
    • Iberian 17.8%
    • Balkan 7.1%

Gedmatch T857316
Gedcom 8884638

Positive result. Pathogenic variant identified in MSH6.
Variant of Uncertain Significance identified in BRCA1.

Previous analysis performed at a different laboratory reportedly identified a variant in MSH6, p.Q1048*, currently known asc.3142C>T (p.Gln1048*), in this individual’s family member. This variant is detected in this individual and is classified as a Pathogenic variant at Invitae.

  • * The MSH6 gene is associated with autosomal dominant Lynch syndrome (also called hereditary nonpolyposis colorectal cancer syndrome, or HNPCC) (MedGen UID: 318886) and autosomal recessive constitutional mismatch repair deficiency syndrome (CMMR-D) (MedGen UID: 78553).
    • This result is consistent with a predisposition to, or diagnosis of, autosomal dominant MSH6-related conditions.
    • Lynch syndrome is characterized by an increased risk of colorectal cancer as well as cancers of the endometrium, ovary, stomach, small intestine, hepatobiliary tract, urinary tract, and brain (PMID: 8550246, 20533284). The estimated lifetime risk of endometrial cancer in women with a Pathogenic MSH6 variant is 16-26% (PMID: 21642682, 20028993) with one study reporting up to 71% (PMID: 15236168). Other lifetime cancer risks include up to 20% for colorectal cancer, up to 13% of gastric cancer, and up to 8% of ovarian cancer (PMID: 23091106, 15236168, 22619739, 20028993, 25070057). Estimated lifetime risks for men include up to 44% risk of colon cancer, up to 30% risk of prostate cancer, and up to 13% risk of gastric cancer (PMID: 20028993, 25070057, 22619739, 23530095). Risks for other types of cancers may also be elevated (PMID: 19861671, 25070057, 23408351, 15740628, 22476430, 22883484). Clinical management guidelines for Lynch syndrome can be found at
    • Close relatives (children, siblings, and each parent) have up to a 50% chance of being a carrier of this variant. More distant relatives may also be carriers. Carriers are at increased risk of developing autosomal dominant MSH6-related conditions and may have reproductive risks related to autosomal recessive MSH6-related conditions as well. Testing for this variant is available.
    • Previous analysis performed at a different laboratory reportedly identified a variant in BRCA1,c.3383T>C (p.Leu1128Pro), in this individual. This variant is detected in this individual and is classified as a Variant of Uncertain Significance at Invitae.
    • The BRCA1 gene is associated with autosomal dominant hereditary breast and ovarian cancer (HBOC) syndrome (MedGen UID: 151793). • The clinical significance of this variant is uncertain at this time. Until this uncertainty can be resolved, caution should be exercised before using this result to inform clinical management decisions